Written by Joyce Smith, BS. Results of two prospective cohort studies and a meta-analysis of 26 cohort studies provide evidence of an inverse association between fruit and vegetable consumption and mortality.
Written by Joyce Smith, BS. This study demonstrates that commonly used nonnutritive sweeteners have the ability to promote the spread of antibiotic-resistant genes in the intestine.
Written by Joyce Smith, BS. This study found that higher leisure time physical activity was associated with reduced major adverse cardiovascular events (MACE) and all-cause mortality risk, while higher occupational physical activity was associated with increased risk, independent of each other.
Written by Joyce Smith, BS. An analyses of phenotype and genes suggests that long-term heavy coffee drinkers with elevated LDL cholesterol may have an increased risk for cardiovascular disease (CVD).
Written by Joyce Smith, BS. This study found no correlation between dietary gluten intake and the risk for incidence Crohn’s disease or ulcerative colitis among women and men without celiac disease.
Written by Angeline A. De Leon, Staff Writer. This study suggests that a specific amino acid (AA) profile is associated with greater bone mineral density (BMD) and lower subsequent fracture risk, independent of diet and lifestyle factors.
Written by Angeline A. De Leon, Staff Writer. An oleuropein-rich extract, with its antiapoptotic, anti-inflammatory, and antioxidant properties, significantly reduced cadmium-induced toxicity in the liver cells of mice.
Written by Joyce Smith, BS. Weight loss and sports supplements containing deterenol have been associated with serious adverse events including cardiac arrest.
Written by Joyce Smith, BS. This study demonstrates how limiting consumption of ultra-processed foods may be an effective strategy for obesity prevention and treatment.
Written by Joyce Smith, BS. Researchers found that the SARS-CoV-2 receptor preferentially recognizes the blood group A antigen expressed on respiratory epithelial cells which may explain why individuals with group A blood may be more susceptible to respiratory failure from COVID-19 infections.
